MOTS-c

Also known as: Mitochondrial ORF of the 12S rRNA type-c

A mitochondrial-derived peptide that regulates metabolism and exercise response.

Overview

MOTS-c is a peptide encoded within the mitochondrial genome, representing a new class of signaling molecules. It regulates metabolic homeostasis and has been called an 'exercise mimetic'.

Mechanism of Action

Activates AMPK pathway, promoting glucose uptake and fatty acid oxidation. Translocates to nucleus to regulate gene expression. Enhances cellular stress resistance.

Pharmacokinetics

Levels decline with age. Exogenous administration shows systemic distribution. Mechanism of cellular entry under investigation.

Dosing Protocols

Note: These are research protocols from literature. This is not medical advice.

1

Research

Dose

5-10 mg

Frequency

3-5x weekly

Duration

4-8 weeks

Based on preclinical extrapolation

Stacking Recommendations

Peptides that may be combined based on complementary mechanisms in research settings.

Rationale: Mitochondrial optimization

Synergy: Different mitochondrial targets

Rationale: Longevity

Synergy: Metabolic and cellular aging

Research Areas

Metabolic SyndromeAgingExercise PhysiologyObesityDiabetes

Key Research Findings

  • 1Improved glucose tolerance in mouse obesity models
  • 2Enhanced exercise capacity and endurance
  • 3Levels correlate with metabolic health in humans
  • 4May mediate some benefits of exercise

Side Effects & Contraindications

Reported Side Effects

  • Limited data available

Contraindications

  • Pregnancy (unknown effects)

Safety Considerations

Limited human safety data. Preclinical studies suggest good tolerability.

Storage Requirements

Store lyophilized at -20C

Scientific References

Quick Reference

Sequence
MRWQEMGYIFYPRKLR
Molecular Weight
2174 g/mol
Half-Life
Not fully characterized
Bioavailability
High (SC)
Research Stage
preclinical
Administration
Subcutaneous injection